Complex Cyanotic Congenital Heart Disease

Patients with complex cyanotic congenital heart disease have arterial oxygen de-saturation resulting from the shunting of systemic venous blood to the arterial circulation, or from cardiac anatomy, which mandates mixing of systemic and pulmonary venous return. The shunting in the circulatory system can occur at the level of the atrium (ASD), the ventricle (VSD), or the great vessels (PDA or aortopulmonary window).

The angiocardiographic, clinical and surgical findings in a complex cyanotic child with dextrocardia, L-loop, laterally inverted but otherwise normally related great arteries and situs ambiguus are described and compared with other similar cases. Complex cyanotic congenital heart disease is the mal formation of the heart, usually of the large blood vessel. The word congenital stand for a situation present at birth; so congenital heart disease means a heart's defect already present at the onset of birth. Since this kind of heart disorder is already present the moment a human life is born, the condition is most prevalent, if not exclusive, among new born babies. Defects affecting the child heart are actually the most ordinary birth defects, causing most of the inborn abnormality-related premature death cases. Just like most defects affecting the infant heart, inborn heart disorder is an abnormality to the flow of the blood. It may be in the form of obstruction or incorrect flow pattern.

Of all possible congenital abnormalities, inborn heart defect accounts for eight in every 1,000 cases, close to 1% of newborn babies. A major factor of this disorder being congenital is genetic predisposition. Genetic influence accounts for the 5 percent of all children with inborn heart defects. Genetic congenital diseases such as Apert syndrome, Conradi syndrome, Crouzon syndrome, Carpenter syndrome, Cornelia de Lange syndrome, and cutis laxa are identified by medical research and studies to increase the risk of having inborn heart defects. Disorders in the chromosomes also significantly contribute as high risk factors of complex cyanotic congenital heart defects. This factor is said to account for the 3% of infants with congenital heart defects. Down's syndrome is a chromosomal disorder leading to congenital heart defects. Roughly almost half of children having Down's syndrome also suffer from inborn heart disorders. Patau syndrome and Edwards's syndrome are also type of chromosomal defects known to be high risks factors of congenital complex heart disorders. Turner syndrome is a disorder in the sex chromosome places 40 percent risk rate of congenital heart defects.

Although it is lower risk level than the other disorders, environmental components are also contributory to risks of inborn heart defects. German measles and acute alcoholism of pregnant women can lead to their babies being born with heart defects. It may be a great wonder for most of the people why pre-birth detection cannot be done to diagnose congenital heart defects. This is so because the fetuses' blood circulation inside the mother's womb is different from how it is after birth. The point is that the difference of blood flow leads to bringing to light the inborn heart defects. Normally the congenital heart disorders are only detected once symptoms like shortness of breath, skin discoloration, fainting, dizziness, inferior appetite, and poor growth are observed.